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CAS: | 89778-27-8 | Synonyms: | FC 1157a; Fareston |
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NK 622: | NSC 613680 | Molecular Formula: | C26H28ClNO.C6H8O7 |
Molecular Weight: | 598.09 | Melting Point:: | 158-164 ºC |
Appearance: | White Or Almost White Powder | ||
High Light: | Anti Estrogen Drugs Bodybuilding,anti estrogen bodybuilding |
99% Antiestrogen Steroid Hormones Toremifene Citrate for Cancer Patients
Description:
Toremifene Citrate is the chemical name of active ingredient in Fareston. Toremifene citrate is very similar to Nolvadex (Tamoxifen Citrate) and Clomid (Clomiphene Citrate), Fareston is medically used to treat breast cancer in post-menopausal women. Toremifene Citrate is also used in many performance enhancing circles for side effect protection and post cycle therapy plans. This does not make Fareston an anabolic steroid; however, it is commonly used by steroid users in order to enhance the safety and efficiency of their steroid use. Anabolic steroid users often use Fareston in order to prevent estrogenic related side effects that may be caused by the use of aromatizing steroids. The most common estrogenic issues include gynecomastia and water retention. High blood pressure can also occur when water retention gets out of hand, but Toremifene Citrate can help with all of these things
Applications:
Toremifene citrate is an oral selective estrogen receptor modulator (SERM) which helps oppose the actions of estrogen in the body. Licensed in the United States under the brand name Fareston, toremifene citrate is FDA-approved for use in advanced (metastatic) breast cancer. It is also being evaluated for prevention of prostate cancer under the brand name Acapodene.
It has already been covered that Toremifene is a SERM, and serves to block Estrogen at various receptor sites in certain tissues within the body (breast tissue in particular). As a layman explanation, Toremifene pretends to be a 'fake' Estrogen that occupies Estrogen receptors within breast tissue. With these receptors occupied by Toremifene, real Estrogen cannot perform their jobs there.
Toremifene does not reduce total blood plasma levels of Estrogen. In addition to being antagonistic to Estrogen receptors in breast tissue, it is also antagonistic to Estrogen at the hypothalamus gland (this essentially 'tricks' the hypothalamus into thinking there is little or no circulating Estrogen levels in the body, causing it to increase its manufacture of Testosterone so that it can utilize aromatization to restore these levels.
Toremifene is also agonistic to Estrogen receptors in other tissues in the body (within the liver in particular). This means that whileToremifene will act as an anti-estrogen in breast tissue and the hypothalamus, it will act as an Estrogen within the liver. This can have beneficial impacts especially during an anabolic steroid cycle, such as improving and shifting cholesterol levels into a more favorable range.
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